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MET inhibitors for targeted therapy of EGFR TKI-resistant lung cancer |  Journal of Hematology & Oncology | Full Text
MET inhibitors for targeted therapy of EGFR TKI-resistant lung cancer | Journal of Hematology & Oncology | Full Text

Resistance to epidermal growth factor receptor inhibition in non-small cell  lung cancer
Resistance to epidermal growth factor receptor inhibition in non-small cell lung cancer

Novel Therapeutic Inhibitors of the c-Met Signaling Pathway in Cancer |  Clinical Cancer Research
Novel Therapeutic Inhibitors of the c-Met Signaling Pathway in Cancer | Clinical Cancer Research

The Role of EGFR-Met Interactions in the Pathogenesis of Glioblastoma and  Resistance to Treatment | Bentham Science
The Role of EGFR-Met Interactions in the Pathogenesis of Glioblastoma and Resistance to Treatment | Bentham Science

Impact of MET alterations on targeted therapy with EGFR-tyrosine kinase  inhibitors for EGFR-mutant lung cancer | Biomarker Research | Full Text
Impact of MET alterations on targeted therapy with EGFR-tyrosine kinase inhibitors for EGFR-mutant lung cancer | Biomarker Research | Full Text

MET/HGF pathway activation as a paradigm of resistance to targeted  therapies - Ko - Annals of Translational Medicine
MET/HGF pathway activation as a paradigm of resistance to targeted therapies - Ko - Annals of Translational Medicine

HER3 and mutant EGFR meet MET | Nature Medicine
HER3 and mutant EGFR meet MET | Nature Medicine

Does c-Met remain a rational target for therapy in patients with EGFR  TKI-resistant non-small cell lung cancer? - Cancer Treatment Reviews
Does c-Met remain a rational target for therapy in patients with EGFR TKI-resistant non-small cell lung cancer? - Cancer Treatment Reviews

Mechanisms of resistance to EGFR tyrosine kinase inhibitors - ScienceDirect
Mechanisms of resistance to EGFR tyrosine kinase inhibitors - ScienceDirect

MET amplification results in heterogeneous responses to osimertinib in EGFR‐mutant  lung cancer treated with erlotinib - Nishiyama - 2020 - Cancer Science -  Wiley Online Library
MET amplification results in heterogeneous responses to osimertinib in EGFR‐mutant lung cancer treated with erlotinib - Nishiyama - 2020 - Cancer Science - Wiley Online Library

Does c-Met remain a rational target for therapy in patients with EGFR  TKI-resistant non-small cell lung cancer? - Cancer Treatment Reviews
Does c-Met remain a rational target for therapy in patients with EGFR TKI-resistant non-small cell lung cancer? - Cancer Treatment Reviews

Phase I results of S49076 plus gefitinib in patients with EGFR  TKI-resistant non-small cell lung cancer harbouring MET/AXL dysregulation -  Lung Cancer
Phase I results of S49076 plus gefitinib in patients with EGFR TKI-resistant non-small cell lung cancer harbouring MET/AXL dysregulation - Lung Cancer

Glioblastoma Cell Resistance to EGFR and MET Inhibition Can Be Overcome via  Blockade of FGFR-SPRY2 Bypass Signaling - ScienceDirect
Glioblastoma Cell Resistance to EGFR and MET Inhibition Can Be Overcome via Blockade of FGFR-SPRY2 Bypass Signaling - ScienceDirect

Cross-talk between MET and EGFR in non-small cell lung cancer involves  miR-27a and Sprouty2 | PNAS
Cross-talk between MET and EGFR in non-small cell lung cancer involves miR-27a and Sprouty2 | PNAS

Figure 1 | Mechanisms of Resistance to EGFR TKIs and Development of a New  Generation of Drugs in Non-Small-Cell Lung Cancer
Figure 1 | Mechanisms of Resistance to EGFR TKIs and Development of a New Generation of Drugs in Non-Small-Cell Lung Cancer

PLOS ONE: Contribution of EGFR and ErbB-3 Heterodimerization to the EGFR  Mutation-Induced Gefitinib- and Erlotinib-Resistance in Non-Small-Cell Lung  Carcinoma Treatments
PLOS ONE: Contribution of EGFR and ErbB-3 Heterodimerization to the EGFR Mutation-Induced Gefitinib- and Erlotinib-Resistance in Non-Small-Cell Lung Carcinoma Treatments

PDF] EGFR and c-Met Inhibitors are Effective in Reducing Tumorigenicity in  Cancer | Semantic Scholar
PDF] EGFR and c-Met Inhibitors are Effective in Reducing Tumorigenicity in Cancer | Semantic Scholar

MET-EGFR dimerization in lung adenocarcinoma is dependent on EGFR mtations  and altered by MET kinase inhibition
MET-EGFR dimerization in lung adenocarcinoma is dependent on EGFR mtations and altered by MET kinase inhibition

Crosstalk between the MET and EGFR pathways. HGF: hepatocyte growth... |  Download Scientific Diagram
Crosstalk between the MET and EGFR pathways. HGF: hepatocyte growth... | Download Scientific Diagram

MET inhibitors for targeted therapy of EGFR TKI-resistant lung cancer |  Journal of Hematology & Oncology | Full Text
MET inhibitors for targeted therapy of EGFR TKI-resistant lung cancer | Journal of Hematology & Oncology | Full Text

Cigarette smoke enhances oncogene addiction to c‐MET and desensitizes EGFR‐expressing  non‐small cell lung cancer to EGFR TKIs - Tu - 2018 - Molecular Oncology -  Wiley Online Library
Cigarette smoke enhances oncogene addiction to c‐MET and desensitizes EGFR‐expressing non‐small cell lung cancer to EGFR TKIs - Tu - 2018 - Molecular Oncology - Wiley Online Library

Dual inhibition of EGFR and c-Met kinase activation by MJ-56 reduces  metastasis of HT29 human colorectal cancer cells
Dual inhibition of EGFR and c-Met kinase activation by MJ-56 reduces metastasis of HT29 human colorectal cancer cells

The Crosstalk of c-MET with Related Receptor Tyrosine Kinases in Urothelial  Bladder Cancer | IntechOpen
The Crosstalk of c-MET with Related Receptor Tyrosine Kinases in Urothelial Bladder Cancer | IntechOpen

Targeting MET as a strategy to overcome crosstalk-related resistance to EGFR  inhibitors - The Lancet Oncology
Targeting MET as a strategy to overcome crosstalk-related resistance to EGFR inhibitors - The Lancet Oncology

Reciprocal and Complementary Role of MET Amplification and EGFR T790M  Mutation in Acquired Resistance to Kinase Inhibitors in Lung Cancer |  Clinical Cancer Research
Reciprocal and Complementary Role of MET Amplification and EGFR T790M Mutation in Acquired Resistance to Kinase Inhibitors in Lung Cancer | Clinical Cancer Research